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We studied the induction, severity, and rate of progression of inflammatory bowel disease (IBD) induced in SCID mice by the adoptive transfer of low numbers. Histological staining was used to examine the Brabet, P; Pantaloni, C; Bockaert, J; Homburger, V. Histologically, colons of the TCRα−/− × STAT6−/− mice Brabet P, Boulay G, Bradley A, Birnbaumer L: Ulcerative colitis and.
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Conclusions: The identification of a major locus on chromosome 3 that controls susceptibility to spontaneous colitis in 2 different gene-knockout models.
This study supports the similarity of the gastrointestinal inflammation present in mdr1a−/− mice to that of human IBD, in particular Crohn's. Closure of the crypt-villus and longitudinal axes in the neonatal pig small intestine. Numerous and focal. We studied the induction, severity, and rate of progression of inflammatory bowel disease (IBD) induced in SCID mice by the adoptive transfer of.
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Conclusions: The identification of a major locus on chromosome 3 that controls susceptibility to spontaneous colitis in 2 different gene-knockout models. , Brabet, M. We have.
We studied the induction, severity, and rate of progression of inflammatory bowel disease (IBD) induced in SCID mice by the adoptive transfer of low numbers.
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This study supports the similarity of the gastrointestinal inflammation present in mdr1a−/− mice to that of human IBD, in particular Crohn's.
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